Cholesterol :: The ABCs of getting rid of excess cholesterol
Excess free cholesterol that accumulates along the walls of blood vessels is transported to the liver for excretion via a process known as reverse cholesterol transport.
Excess free cholesterol that accumulates along the walls of blood vessels is transported to the liver for excretion via a process known as reverse cholesterol transport.
In animals and humans, the proteins known as liver X receptors (LXRs) sense cholesterol levels. Upon sensing increases in cholesterol, these receptors activate signaling pathways that remove diet-derived cholesterol from the body.
Got high cholesterol? You might want to stay away from air pollution. That?s the message of a new UCLA study linking diesel exhaust to atherosclerosis, or hardening of the arteries, which significantly increases one?s risk for heart attack and stroke.
Should we be watching our exposure to airborne pollution as well as our cholesterol levels” Research now indicates that air pollution has a role to play in atherosclerosis (artery hardening), which can contribute to heart attacks or strokes.
One route to a long and healthy life may be establishing the right balance in insulin signaling between the brain and the rest of the body, according to new research from Children’s Hospital Boston.
Results from two studies indicate that elevated nonfasting triglyceride levels are associated with cardiovascular events such as a heart attack, with one study finding that triglyceride levels measured after fasting does not show this association, according to studies in the July 18 issue of JAMA.
Orange juice, despite its high caloric load of sugars, appears to be a healthy food for diabetics due to its mother lode of flavonoids, a study by endocrinologists at the University at Buffalo has shown.
Current PET-CT scanners with standard commercial software designed to provide images of the heart are falsely indicating coronary artery disease in as many as 40 percent of patients, according to a study published today on the cover page of the Journal of Nuclear Medicine.
We?ve all wondered how a seemingly healthy person can actually be at high risk for heart disease or a heart attack. Now researchers have uncovered a new clue to this mystery. The culprit: myeloperoxidase (MPO), a protein secreted by white blood cells that both signals inflammation and releases a bleach-like substance that damages the cardiovascular system.
People who have a reduced lung capacity may have a greater risk of heart attack and stroke because they show evidence of inflammation, reveals a study published online ahead of print in Thorax.