Aging :: DNA damage mimics aging
The accumulation of genetic damage in our cells is a major contributor to how we age, according to a study being published today in the journal Nature by an international group of researchers.
The accumulation of genetic damage in our cells is a major contributor to how we age, according to a study being published today in the journal Nature by an international group of researchers.
The surge of baby boomers now entering their 60s means more drivers on the road who may be impaired by dementia or other cognitive impairments linked to aging. Researchers at the Alzheimer’s Disease Research Center (ADRC) of Washington University School of Medicine in St. Louis and elsewhere have developed a three-hour workshop that trains health care providers to identify potentially unsafe drivers with dementia and to encourage appropriate retirement from driving.
A new imaging molecule that can detect and map plaques and tangles in the brains of people with Alzheimer’s disease could eventually lead to earlier diagnosis of the devastating disease, researchers at the University of California, Los Angeles report in the Dec. 21, 2006, issue of the New England Journal of Medicine.
For older Americans, reversal of the brain shrinkage that occurs as people age is just one benefit of greater physical activity, according to research published in the latest issue of the Journal of Gerontology — Medical Sciences (Vol. 61A, No. 11). In a special section devoted to exercising and aging, this edition of the journal features five separate reports on the topic.
University of Arkansas public radio station KUAF 91.3 FM has been awarded a $15,000 grant from the American Psychiatric Foundation for the production of a year-long five-part series of in-depth reports on mental health and the elderly.
Memory training and other non-drug treatments may one day help older adults ward off declines in mental function, according to researchers from Wake Forest University School of Medicine in an editorial in the current issue of the Journal of the American Medical Association.
Researchers at the University of Southern California have discovered a direct link between loss of testosterone and the development of an Alzheimer’s-like disease in mice. They also discovered that testosterone treatment slows progression of the disease.The study, published in the Dec. 20 issue of the Journal of Neuroscience, predicts that testosterone-based hormone therapy may be useful in the treatment and prevention of Alzheimer?s disease in aging men.
Waning testosterone levels in aging men may be responsible for a slew of physical symptoms, dementia among them. Recent studies indicate that men with Alzheimer”s disease (AD) have lower testosterone level than aged men without AD.
Experiments on mouse models of Alzheimer’s disease suggest that treatment with male sex hormones might slow its progression. The findings, published in the Dec. 20 issue of the Journal of Neuroscience, provide new insight into the relationship between testosterone loss and AD, which affects 4.5 million Americans.
Results of a new study in PLoS Medicine provide the first indications that application of hyaluronate to atrophic skin might be useful therapeutically and suggest that clinical investigation is warranted.